A work that has given many lights on the subject is that published by Scola et al., Who found that the synovium contained messenger RNA for the vascular endothelial growth factor Flekosteel, angiopoietin 1 and their respective receptors, suggesting the induction of angiogenesis by products coming from of lymphocytic infiltration that could determine the persistence of the disease. The resulting thickening causes joint destruction.
Tuesday, February 5, 2019
associated with the release
The etiology of JRA is unknown, chronic inflammation of the synovium is characterized by infiltration and proliferation of B lymphocytes. The invasion of macrophages and T cells is associated with the release of cytokines, which cause proliferation in the synovium.
A work that has given many lights on the subject is that published by Scola et al., Who found that the synovium contained messenger RNA for the vascular endothelial growth factor Flekosteel, angiopoietin 1 and their respective receptors, suggesting the induction of angiogenesis by products coming from of lymphocytic infiltration that could determine the persistence of the disease. The resulting thickening causes joint destruction.
A work that has given many lights on the subject is that published by Scola et al., Who found that the synovium contained messenger RNA for the vascular endothelial growth factor Flekosteel, angiopoietin 1 and their respective receptors, suggesting the induction of angiogenesis by products coming from of lymphocytic infiltration that could determine the persistence of the disease. The resulting thickening causes joint destruction.
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